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TRIlogy-5 Multiple myeloma
A Phase 3, randomized, open-label study comparing ramantamig (JNJ-79635322) and teclistamab in patients with relapsed or refractory MM who have received 1–3 prior lines of therapy
Phase
Compound
Ramantamig (JNJ-79635322; BCMA × GPRC5D × CD3 trispecific Ab) or teclistamab (BCMA × CD3 bispecific Ab)
NCT identifier
NCT07518186
Enrollment

N=~700

For more information, including
contact details, please visit
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Study overview & design

Key eligibility criteria*:

  • Relapsed or refractory MM
  • 1–3 prior lines of therapy, including anti-CD38 Ab and lenalidomide
  • ECOG PS of 0–2
  • No known active or prior CNS involvement or clinical signs of meningeal involvement of MM
Study design
Study design

Primary outcomes:

  • ≥CR
  • PFS

Secondary outcomes:

  • ORR
  • ≥VGPR
  • DOR
  • MRD-negative CR
  • MRD-negative CR at 1 year and at 5 years
  • OS
  • TTNT
  • Safety
  • HRQoL

*Not a complete list of inclusion and exclusion criteria. Not a complete list of outcomes.

Ab, antibody; BCMA, B-cell maturation antigen; CD, cluster of differentiation; CNS, central nervous system; CR, complete response; DOR, duration of response; ECOG PS, Eastern Cooperative Oncology Group performance status; GPRC5D, G protein–coupled receptor class C group 5 member D; HRQoL, health-related quality of life; MM, multiple myeloma; MRD, minimal residual disease; ORR, overall response rate; OS, overall survival; PD, progressive disease; PFS, progression-free survival; R, randomized; SC, subcutaneous; TTNT, time to next line of therapy; VGPR, very good partial response.

Recruiting site locations
World Map
Israel, Taiwan, Turkey, United States
Study design
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Ramantamig mechanism of action
The following is video of the proposed mechanism of action for ramantamig, an investigational product which has not been reviewed by the FDA. Correlation to clinical effect has not been established.
Ramantamig mechanism of action
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Mechanism of disease in multiple myeloma
Mechanism of disease in multiple myeloma
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